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This concern was further highlighted by an analysis of the US FDA adverse events database

Presented at: American Academy of Neurology Annual Meeting

While mechanistic studies suggest that VEs antioxidant properties attenuate lipid peroxidation cascades and mitochondrial dysfunction, emerging evidence positions VE as a pleiotropic regulator, suppressing glutamate excitotoxicity, stabilizing BBB integrity, and tempering IL-1-mediated neuroinflammation (Fig