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Ketamine and psilocybin each produce a brief acute experience but set off a wave of synaptic growth afterward, ketamine through glutamate release and mTOR-dependent synapse formation and psilocybin through rapid, lasting growth of dendritic spines in the frontal cortex, both via BDNF signaling, which is why a single session can shift symptoms for weeks

Its GHS-R1a selectivity delivers a GH pulse equivalent to GHRP-6 or Hexarelin without the off-target effects (cortisol, prolactin, hunger)

No EMA (European Medicines Agency) or MHRA approval exists for any indication

For example, 10 mg reconstituted in 2 mL gives 5 mg/mL, which is the same as 500 mcg per 0.1 mL