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When -glutamyl- S -allyl-L-cysteine was used as a -glutamyl donor substrate, S -allyl-L-cysteine was not formed at a detectable level in assays using crude protein extracts from control yeast ( Figure 3

Using a 1:1 propensity score matching (PSM) model, we matched patients of both cohorts based on baseline demographics, comorbidities (hypertensive disorders, ischemic heart disease, gallstones, annular pancreas, alcohol use disorders, hypertriglyceridemia, hypercalcemia, cystic fibrosis, and cannabis use), medications known to cause drug-related pancreatitis, and laboratory values

A study conducted using a rat model of type 1 diabetes demonstrated reduced inflammatory cell infiltration and decreased glomerular expression of inflammatory markers, including TGF-1 and intercellular adhesion molecule-1, with administration of exendin-4 [51]

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