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In multiple sclerosis models, GLP-1 receptor activation reduces pathogenic Th1 and Th17 cell activity, inhibits microglial activation (the brain equivalent of macrophage inflammation), and may enhance remyelination processes
There has been modeling done to explore alternative dosing regimens of GLP-1s that simultaneously reduce costs and maintain weight-loss efficacy, but theres limited evidence and no formal guidelines to support this practice, Zimmermann says
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