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established decades ago that meaningful weight loss universally involves some reduction in fat-free mass (Chaston et al

Collectively, these findings establish a mechanistic pathway whereby LEO restores dysregulated SCFA production in diabetic mice, highlighting dose-responsive butyrate accumulation as the dominant effector mediating enhanced GLP-1 secretion in functionally compromised enteroendocrine cells

These results combined with selective activation of G s by OA suggest that stimulation of PI hydrolysis is independent of PKA, and probably involves activation of the cAMP-dependent Epac/PLC- pathway

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