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ALCAR limits PCa induced pro-angiogenic features and regulates CXCL8-CXCR1/2, CCL2-CCR2 and CXCL12-CXCR4 axis in endothelial cells The vascular endothelial growth factor (VEGF) and CXCL8, a CXC inflammatory chemokine, have been demonstrated to induce angiogenesis and promote progression of PCa [44, 45]

In this episode we cover: WHY MOST TRAINEES SELECT LOADS THAT ARE TOO LIGHT RESEARCH SHOWING LIFTERS CHOOSE ~53% OF THEIR 1RM WHEN SELF-SELECTING WEIGHTS WHY LOW-LOAD TRAINING REQUIRES PUSHING CLOSER TO FAILURE TO STIMULATE HYPERTROPHY THE LIMITATIONS OF USING RIR AND HOW INACCURATE FAILURE PREDICTION CAN BE WHY MANY LIFTERS BELIEVE THEYRE TRAINING TO FAILURE WHEN THEY ARENT WHY META-ANALYSES SHOW A DOSE-RESPONSE RELATIONSHIP BETWEEN VOLUME AND MUSCLE GROWTH THE PELLAND META-REGRESSION AND WHY HIGHER VOLUMES DONT SHOW A CLEAR INVERTED U HOW EFFORT PER SET MAY CHANGE AS TRAINING VOLUME INCREASES BEHAVIORAL CHANGES THAT MAY OCCUR DURING HIGH-VOLUME TRAINING SESSIONS SANDBAGGING, PACING, AND KEEPING REPS IN RESERVE THE POTENTIAL ROLE OF THE CENTRAL GOVERNOR THEORY IN REGULATING EFFORT WHY RPE OFTEN TOPS OUT AROUND 7-8 IN MANY TRAINING STUDIES WHETHER THIS REFLECTS EFFORT REGULATION OR ADAPTATION TO HIGH WORKLOADS PRACTICAL COACHING INSIGHTS ON HOW TO DETERMINE IF YOURE TRULY TRAINING HARD ENOUGH Ultimately, the goal of this episode is to encourage you to ask yourself one honest question: Am I really training as hard as I think I am

A smaller dose has less side effects, but does it achieve what you are hoping to achieve

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