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914 Mechanistically, TMAO was able to accelerate the progression of atherosclerosis by inducing vascular endothelial cells (VECs) pyroptosis through SDHB/ROS pathway, 15 contribute to endothelial dysfunction by increasing HMGB1 expression and disrupting cell-cell junction proteins, 16 promote vascular calcification by inducing osteogenic differentiation of vascular smooth muscle cells (VSMCs) via NLRP3 inflammasome and NF-B pathway, 17 and exacerbate cardiac function and cardiac fibrosis after MI by promoting the transition of fibroblasts into myofibroblasts through TGF-RI/Smad2 axis

Gaber AO, Oxley D, Karas J et al (1991) Changes in gastric emptying in recipients of successful combined pancreas-kidney transplants

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Schmutzler C, Mentrup B, Schomburg L, Hoang-Vu C, Herzog V, Khrle J (2007) Selenoproteins of the thyroid gland: expression, localization and possible function of glutathione peroxidase 3