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Histopathological examination revealed marked structural alterations and synaptic ultrastructural damage in the hippocampal CA1 region, while proteomic analysis identified differentially expressed proteins enriched in pathways related to synaptic plasticity, neurotransmitter release, and associated signaling cascades (Chuwen et al., 2025)

The study found increased risk in both patients with type 2 diabetes and patients using semaglutide for overweight or obesity, but the authors noted that the study was observational and could not prove that semaglutide directly caused NAION

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