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We then found that PA treatment combined with CD36 overexpression increases mitochondrial ROS production, TfR1 protein expression, and the iron content of TNBC cells overexpressing CD36 compared to the wild-type TNBC

A naturally occurring antioxidant in the body that helps support cellular health, immune function, and detoxification processes

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[9] The proposed mechanism of GHK involves its interaction with the pathways controlling iron release from ferritin, potentially reducing iron release by approximately 87%