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In adipose tissue, PYY (3-36) inhibits lipolysis, promotes lipid storage, and enhances systemic insulin sensitivity by regulating the secretion of fat factors such as adiponectin and leptin, thus improving insulin resistance and glucose homeostasis

This gene modulation explains why GHK-Cu benefits extend across such diverse tissues

Moreover, memory deficits induced by administration of the drug, scopolamine, were effectively reversed by this Rimonabant treatment

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