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Dual inhibition of thioredoxin reductase and proteasome is required for auranofin-induced paraptosis in breast cancer cells

Therefore, we hypothesize that BPC157 may reduce intracellular ROS levels through an antioxidant mechanism similar to that reported in J Clin Invest [19], thereby stabilizing BACH1 protein and enabling its pro-angiogenic function

Chemical oxidants can rapidly decrease intracellular GSH, but are easy to produce toxic side effects such as hemolysis and nephrotoxicity [6,7,8,9]

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